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Int J Clin Exp Pathol 2014;7(2):537551 www.ijcep.com /ISSN:19362625/IJCEPOriginal Post NOX1 is accountable for cell death via STAT3 activation in hyperoxia and is connected with the pathogenesis of Acute Respiratory Distress SyndromeStephanie Carnesecchi1,2, Isabelle DunandSauthier2, Filippo Zanetti1,2, Grigory Singovski1,two, Christine Deffert2, Yves Donati1,2, Thomas Cagarelli1,two, JeanClaude Pache2, KarlHeinz Krause2, Walter Reith2, Constance BarazzoneArgiroffo1,Division of Pediatrics, Geneva, Switzerland; 2Department of Pathology and Immunology, Healthcare School, University of Geneva, SwitzerlandReceived December six, 2013; Accepted December 21, 2013; Epub January 15, 2014; Published February 1, 2014 Abstract: Reactive oxygen species (ROS) contribute to alveolar cell death in Acute Respiratory Distress Syndrome (ARDS) and we previously demonstrated that NOX1derived ROS contributed to hyperoxiainduced alveolar cell death in mice.N-Fmoc-N-(2-phenylethyl)-glycine Purity The study investigates irrespective of whether NOX1 expression is modulated in epithelial cells concomitantly to cell death and related to STAT3 signaling within the exudative phase of ARDS.2212021-56-0 site Moreover, the function of STAT3 activation in NOX1dependent epithelial cell death was confirmed by using a lung epithelial cell line and in mice exposed to hyperoxia.PMID:32695810 NOX1 expression, cell death and STAT3 staining were evaluated inside the lungs of handle and ARDS patients by immunohistochemistry. In parallel, a steady NOX1silenced murine epithelial cell line (MLE12) and NOX1deficient mice had been made use of to characterize signalling pathways. In the present study, we show that NOX1 is detected in alveolar epithelial cells of ARDS individuals within the exudative stage. Furthermore, increased alveolar epithelial cell death and phosphorylated STAT3 are observed in ARDS individuals and linked with NOX1 expression. Phosphorylated STAT3 can also be correlated with TUNEL staining. We also confirmed that NOX1dependent STAT3 activation.